Antibodies against desmoglein 3. Usually starts in the MOUTH — painful erosions that will not heal, often months before any skin blister. Flaccid blisters that rupture into raw areas. The most serious of the pemphigus group.
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Antibodies against desmoglein 1. Superficial, scaly and crusted — scalp, face, chest, upper back. Characteristically SPARES the mouth, which is what separates it from vulgaris.
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A DIFFERENT disease from pemphigus, not a variant of it. Antibodies against BP180/BP230 at the basement membrane, so the split is deeper and the blisters are TENSE, not flaccid. Mostly over 70. The intense itch usually arrives months before the first blister — and is very often treated as eczema or scabies until it does.
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The wider pemphigoid family. Mucous membrane (cicatricial) pemphigoid SCARS — mouth and eyes — and ocular involvement can threaten sight and is an emergency. Pemphigoid gestationis occurs in pregnancy.
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⚠ Inherited EB is GENETIC, not autoimmune: a mutation means the proteins anchoring your skin were never built correctly. No formulation corrects a gene, and we do not claim to. Only epidermolysis bullosa ACQUISITA (EBA) is autoimmune — and that one does belong in this hub.
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